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Furthermore, puscher studies did not examine whether the association differs by beverage type. Brain Res Mol Brain Res ; Risk of injury after alcohol consumption: We conducted stratified analyses to assess whether the association differed by drink type beer, wine, liquordrinking patterns every day of the week vs.
Major depressive disorder and hypothalamic-pituitary-adrenal axis activity: Hippocampal and amygdala changes in patients with major depressive disorder and healthy controls during a 1-year follow-up.
Epigenetic and inflammatory marker profiles manua with depression in a community-based epidemiologic sample.
Table 1 summarizes the characteristics of the study population. J Gen Intern Med. Tets subunit immunoreactivity on apical pyramidal cell dendrites in hippocampus of schizophrenics and manic depressives.
The etiology of major depression is complex because involves psychosocial, genetic, and biological factors. Differential effect of chronic antidepressant treatments on lipopolysaccharideinduced depressive like behavioural symptoms in the rat. These long-lasting changes produce variations at the structural level and in the functioning of different brain areas.
BDNF has opposite effects on the quantal amplitude of pyramidal neuron and interneuron excitatory synapses. Hippocampal morphology and distinguishing late-onset from early-onset elderly depression. Remodeling of hippocampal spine synapses in the rat learned helplessness model of depression. Recent heavy drinking of alcohol and embolic stroke.
Five cases of interferon-alpha-induced depression treated with antidepressant therapy.
Statistical Analysis We applied standard methods for stratified data analysis, where the stratifying variable is the individual participant. We did not have enough data to further stratify these subgroups to determine whether other participant characteristics were responsible for the heterogeneity in the observed RRs. Brain-derived neurotrophic factor as a biomarker for mood disorders: The neurobiology of stress: Hippocampal changes and white matter lesions in early-onset depression.
Abstract Major depression represents a public health problem due to its high prevalence. Cellular and molecular mechanisms in the long-term action of antidepressants.
In a sensitivity analysis excluding individuals also exposed to other potential risk factors, our findings were not materially altered.
Trigger descarga and their attributable risk for rupture of intracranial aneurysms: Finally, our results were similar when we excluded participants who were interviewed more than a week after AMI onset. Genetic risk factors for major depression in men and women: Activation of central nervous system inflammatory pathways by interfer-on-alpha: Brain volume abnormalities in major depressive disorder: Plasma concentrations of gamma-aminobutyric acid GABA and mood disorders: Adding psychotherapy to pharmacotherapy in the mannual of depressive disorders in adults: Serotonin transporter gene polymorphism and affective disorder.
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Patterns of alcohol consumption and ischaemic heart disease in culturally divergent countries: Despite years of study, the biological basis of major depression and precise mechanisms of antidepressant efficacy remain unclear.
Recent alcohol consumption, cigarette smoking, and cerebral infarction in young adults.
lusched We estimated the proportion of cases whose alcohol consumption was associated with AMI onset in the following hour using the formula for the population attributable fraction. The elevated risk in the first hour was followed by reduced risk in the next 23 hours, producing an overall protective association for beer and wine in the hour window before MI.
The publisher’s final edited version of this article is available at Epidemiology.
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